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378397 in the activation segment, including salt bridges between Glu379 from one protomer and Arg216 and Arg241 of the other protomer 8

Wang Y, Singh R, Massey AC, Kane SS, Kaushik S, Grant T, et al

While these reports highlight lower circulating glycine as an emerging biomarker for both NAFLD/NASH and ASCVD, studies in humans and mice support a causative role of reduced glycine availability and the potential of glycine-based treatment in both diseases (199, 201)

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kunkeei (Supplementary Table S5: Spearmans , r s = 0.81, p = 0.0002), but was explained by both age and ontogeny